IgA Nephropathy And Tonsil Resection

One major problem for IgA nephropathy patients is recurrent hematuria (blood in urine) with or without proteinuria. Hematuria can be gross (blood can be seen with naked eyes) or microscopic (blood can only be seen under microscope).

Often IgA nephropathy patients will have upper respiratory tract infections before the onset of hematuria such as tonsillitis. Effective prevention and control of respiratory tract infections and tonsillitis can help reduce the risk of blood in urine. For those that have repeated tonsillitis, tonsil resection can be done under the doctor’s permission and guidance. It has been proved to help relieve the onset of hematuria and proteinuria and it has long-term protection of kidney functions.

Post-surgery nursing for IgA nephropathy patients are very important. After tonsil resection, patients can have the complications of bleeding and infections, therefore effective prevention and proper measures should be had to prevent further harms to the kidney functions and other parts of the body.

For IgA nephropathy patients, there are other treatment options if you do not want to have the tonsil resection. Since it is an auto-immune kidney disease, immunotherapy is a good treatment because it can help regulate immune disorders and repair damaged renal tissues so as to relieve symptoms and improve renal functions. When renal functions are improved, the kidneys can prevent the leaking of protein and red blood cells into urine, remove excess water and wastes from the blood. When the immune function is improved, patients will be less susceptible to tonsillitis and other infections, therefore the risk factors for IgA nephropathy patients will be much reduced.

Besides, IgA nephropathy patients should try to avoid cold, over-tiredness and the use of nephrotoxicity drugs. Regular practicing of some mild and gentle exercises such as tai chi and qi gong can help enhance the patient’s immunity and disease-resistance ability so as to help reduce the chance of infections and inflammations which are common inducements of IgA nephropathy relapse and the onset of blood and protein in urine, swelling and high blood pressure, etc.